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However, the mechanisms are complex, and it seems like salsolinol would reduce GABAergic activity while ethanol increases it, suggesting opposite responses on GABAergic receptor activity from ethanol and one of its metabolites, also causing a downstream opposite effect on dopamine release (Peana et al., 2017)

[195] [212] This internalization is mediated by RhoA activation and its downstream effector ROCK, a process that is TAAR1-dependent